Search results for "Neuromuscular Blocking Agents"

showing 5 items of 5 documents

Perioperative nonspecific histamine release : a new classification by aetiological mechanisms and evaluation of their clinical relevance

1993

As a consequence of the performance of a randomized controlled clinical trial on perioperative histamine release and cardiovascular and respiratory disturbances, several types of increases in plasma histamine had to be distinguished instead of only two which existed at the beginning of the study: drug-induced allergic and pseudoallergic reactions. First of all, the new classification by aetiology (clinical epidemiology) was derived from a meta-analysis (secondary analysis) of the most recent literature. According to that histamine release in the perioperative period has several, different causes and is involved in several, different disease manifestations. A clear distinction (classificatio…

AdultMaleHistamine ReleaseDrug HypersensitivityIntraoperative Periodchemistry.chemical_compoundHumansMedicineAnesthesiaClinical significanceProspective StudiesAgedAnestheticsNeurosecretionbusiness.industryGeneral MedicinePerioperativeMiddle AgedPathophysiologyAnalgesics OpioidClinical trialAnesthesiology and Pain MedicinechemistryAnesthesiaEtiologyGastric acidFemalePremedicationNeuromuscular Blocking AgentsbusinessHistamineHistamineAnnales Françaises d'Anesthésie et de Réanimation
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Sugammadex, a Neuromuscular Blockade Reversal Agent, Causes Neuronal Apoptosis in Primary Cultures

2013

Sugammadex, a γ-cyclodextrin that encapsulates selectively steroidal neuromuscular blocking agents, such as rocuronium or vecuronium, has changed the face of clinical neuromuscular pharmacology. Sugammadex allows a rapid reversal of muscle paralysis. Sugammadex appears to be safe and well tolerated. Its blood-brain barrier penetration is poor (< 3% in rats), and thus no relevant central nervous toxicity is expected. However the blood brain barrier permeability can be altered under different conditions (i.e. neurodegenerative diseases, trauma, ischemia, infections, or immature nervous system). Using MTT, confocal microscopy, caspase-3 activity, cholesterol quantification and Western-blot we …

Estrès oxidatiuSmac/Diablo and CASP-3.BiologyPharmacologymedicine.disease_causeSugammadexSugammadexAIFmedicineAnimalsRocuroniumCytCCells CulturedFisiologia cel·lularNeuronsNeuromuscular BlockadeapoptosisGeneral MedicineNeuromuscular Blocking AgentsRatsOxidative StressApoptosisAnesthesiaToxicityNeuromuscular BlockadeNeuron deathOxidative stressmedicine.drugResearch Papergamma-CyclodextrinsInternational Journal of Medical Sciences
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Effects of Neuromuscular Blocking Agents on Neuronal Nicotine Receptors of Motor Nerves: Blockade of Nicotinic Autofacilitation and Backfiring

1991

ABSTRACT Tubocurarine reduced (60%) [3H]acetylcholine release from the isolated rat phrenic nerve at concentrations of 1 or 10 μM, but blocked backfiring of the phrenic nerve occuring after partial blockade of cholinesterase already at a concentration of 100 nM. The distinct potency of tubocurarine suggests differences between the presynaptic nicotine receptors mediating autofacilitation of release and preterminal nicotine receptors mediating backfiring of the motor nerve or a low agonist concentration at the latter receptors. The new neuromuscular blocking agents atracurium, pancuronium and vecuronium less effectively reduced [3H]acetylcholine release than tubocurarine indicating a lower a…

NicotineAgonistNicotinic agonistChemistrymedicine.drug_classmedicineMotor nervePharmacologyNeuromuscular Blocking AgentsReceptorAcetylcholinemedicine.drugPhrenic nerve
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Post-anaesthesia pulmonary complications after use of muscle relaxants (POPULAR): a multicentre, prospective observational study

2019

Background: Results from retrospective studies suggest that use of neuromuscular blocking agents during general anaesthesia might be linked to postoperative pulmonary complications. We therefore aimed to assess whether the use of neuromuscular blocking agents is associated with postoperative pulmonary complications. Methods: We did a multicentre, prospective observational cohort study. Patients were recruited from 211 hospitals in 28 European countries. We included patients (aged ≥18 years) who received general anaesthesia for any in-hospital procedure except cardiac surgery. Patient characteristics, surgical and anaesthetic details, and chart review at discharge were prospectively collecte…

Pulmonary and Respiratory Medicinepulmonary complications muscle relaxants Post-anaesthesia complicationsNeuromuscular Blockadepulmonary complication muscle relaxantneuromuscular block ; postoperative pulmonary complicationbusiness.industryRetrospective cohort studypost-operative pulmonary complicationsNeuromuscular monitoringNeuromuscular Blocking AgentsSugammadexNOAnaesthesia03 medical and health sciences0302 clinical medicine030228 respiratory systemAnesthesiaMedicineGeneral anaesthesiaNeuromuscular Agents030212 general & internal medicineMED/41 - ANESTESIOLOGIAProspective cohort studybusinessmedicine.drug
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Neuronal effects of Sugammadex in combination with Rocuronium or Vecuronium.

2017

Rocuronium (ROC) and Vecuronium (VEC) are the most currently used steroidal non-depolarizing neuromuscular blocking (MNB) agents. Sugammadex (SUG) rapidly reverses steroidal NMB agents after anaesthesia. The present study was conducted in order to evaluate neuronal effects of SUG alone and in combination with both ROC and VEC. Using MTT, CASP-3 activity and Western-blot we determined the toxicity of SUG, ROC or VEC in neurons in primary culture. SUG induces apoptosis/necrosis in neurons in primary culture and increases cytochrome C (CytC), apoptosis-inducing factor (AIF), Smac/Diablo and Caspase 3 (CASP-3) protein expression. Our results also demonstrated that both ROC and VEC prevent these…

vecuroniumNecrosisEstrès oxidatiuPrimary Cell CulturerocuroniumCaspase 3NeuronesPharmacologySugammadexSugammadex03 medical and health sciences0302 clinical medicine030202 anesthesiologymedicineAnimalsHumansAndrostanolsRocuroniumCell damageNeuronsVecuronium BromideDose-Response Relationship DrugCaspase 3business.industryapoptosis.Apoptosis Inducing FactorCytochromes c030208 emergency & critical care medicineGeneral Medicinemedicine.diseaseneuronRatsDrug Combinationsmedicine.anatomical_structureGene Expression RegulationApoptosisToxicityNeuronNeuromuscular Blocking Agentsmedicine.symptombusinessResearch Papergamma-Cyclodextrinsmedicine.drug
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